NTU Trường Đại học Nha Trang
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Cancer cell., Tập 22, Số 6, 2012
Mục lục:
DòngNội dung
1EMT andMET in Metastasis:Where Arethe Cancer Stem Cells? / Thomas Brabletz
2Disputed Paternity: The Uncertain Ancestryof Pancreatic Ductal Neoplasia / Anirban Maitra, Steven D. Leach
3Chemokine to the Rescue: Interleukin-8 MediatesResistance to PI3K-Pathway-Targeted Therapyin Breast Cancer / Robert T. Abraham
4A New ‘‘Brew’’ of MALT1 Inhibitors / Ryan M. Young, Louis M. Staudt
5Metastatic Colonization Requires the Repression of the Epithelial-Mesenchymal Transition Inducer Prrx1 / Oscar H. Ocana and others
6Spatiotemporal Regulation of Epithelial-Mesenchymal Transition Is Essential for Squamous Cell Carcinoma Metastasis / Jeff H. Tsai and others
7Identification of Sox9-Dependent Acinar-to-Ductal Reprogramming as the Principal Mechanism for Initiation of Pancreatic Ductal Adenocarcinoma / Janel L. Kopp and others
8Cell-type, Dose, and Mutation-type Specificity Dictate Mutant p53 Functions In Vivo / Ming Kei Lee and others
9The EphA2 Receptor Drives Self-Renewal and Tumorigenicity in Stem-like Tumor-Propagating Cells from Human Glioblastomas / Elena Binda and others
10CRL4B Catalyzes H2AK119 Monoubiquitination and Coordinates with PRC2 to Promote Tumorigenesis / Huili Hu and others
11JAK2/STAT5 Inhibition Circumvents Resistance to PI3K/mTOR Blockade: A Rationale for Cotargeting These Pathways in Metastatic Breast Cancer / Adrian Britschgi and others
12MALT1 Small Molecule Inhibitors Specifically Suppress ABC-DLBCL In Vitro and In Vivo / Lorena Fontan and others
13Pharmacologic Inhibition of MALT1 Protease by Phenothiazines as a Therapeutic Approach for the Treatment of Aggressive ABC-DLBCL / Daniel Nagel and others